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A study from the University of California, Los Angeles suggests creatine may strengthen the immune system's fight against cancer.
Source: University of California, Los Angeles
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The study was published in the journal iScience.
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The study found that creatine boosts the activity of dendritic cells, which are specialized immune cells that detect tumors and activate killer T cells.
Source: University of California, Los Angeles
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The findings were based on experiments in mice and human cells.
Source: University of California, Los Angeles
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The results have not yet been tested in human patients.
Source: University of California, Los Angeles
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Approximately 20% to 40% of patients experience meaningful benefits from current cancer immunotherapies designed to activate killer T cells.
Source: University of California, Los Angeles
Lili Yang, professor of microbiology, immunology and molecular genetics at UCLA
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"Immunotherapy has shown remarkable promise, but it only works for a subset of patients," said Lili Yang, the study's senior author and a professor of microbiology, immunology and molecular genetics at UCLA.
Lili Yang, professor of microbiology, immunology and molecular genetics at UCLA
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"What this study shows is that creatine doesn't just help the T cells fighting cancer -- it also energizes the entire infrastructure supports and guides them," said Lili Yang.
Lili Yang, professor of microbiology, immunology and molecular genetics at UCLA
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"That makes creatine a promising supplement to holistically support the immune response that modern immunotherapies depend on," said Lili Yang.
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Researchers found that the gene responsible for producing the creatine transporter was more active in tumor-infiltrating dendritic cells in mice than in dendritic cells in healthy tissue.
Source: University of California, Los Angeles
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Engineered dendritic cells lacking the creatine transporter survived less effectively, became less active, and were less capable of preparing T cells to recognize and attack tumors in laboratory experiments.
Source: University of California, Los Angeles
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T cells multiplied less and produced fewer signaling molecules when grown alongside creatine-deficient dendritic cells in laboratory experiments.
Source: University of California, Los Angeles
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Daily creatine injections in mouse models of melanoma significantly slowed tumor growth.
Source: University of California, Los Angeles
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Daily creatine injections in mouse models of melanoma increased the number and activity of dendritic cells that had entered tumors.
Source: University of California, Los Angeles
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Treated dendritic cells released higher levels of chemical signals that attracted additional immune cells into the tumor environment in mouse models.
Source: University of California, Los Angeles
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Creatine supplementation increased intracellular ATP levels in dendritic cells, according to metabolomics analyses in the study.
Source: University of California, Los Angeles
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In laboratory experiments, creatine enhanced the activation of human monocyte-derived dendritic cells.
Source: University of California, Los Angeles
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Creatine improved the ability of human monocyte-derived dendritic cells to stimulate human T cells against a cancer-associated target in laboratory experiments.
Source: University of California, Los Angeles
James Elsten-Brown, graduate student
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"The potential we see here is that creatine could be used in two complementary ways: as a supplement to enhance the immune response of patients already receiving immunotherapy, and as a tool to improve the quality of dendritic cell-based vaccines before they're administered," said James Elsten-Brown, a co-first author and graduate student in Yang's lab.
Elliot Kang, former undergraduate student researcher
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"Understanding how to metabolically support dendritic cells is about supporting the entire anti-tumor response, not just the killer T cells at the end of it," said Elliot Kang, a co-first author of the study and former undergraduate student researcher in Yang's lab.
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The experimental approaches described in the study have not been approved by the Food and Drug Administration as safe and effective for use in people.
Source: University of California, Los Angeles
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Funding for the research was provided by a UCLA Broad Stem Cell Research Center Rose Hills Foundation Innovator Grant.
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Funding for the research was provided by the UCLA Health Jonsson Comprehensive Cancer Center and UCLA Broad Stem Cell Research Center Ablon Scholars Program.
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Funding for the research was provided by a Magnolia Council Senior Investigator Grant Award and a fellowship from the Tower Cancer Research Foundation.
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A patent application regarding the potential therapeutic strategy was filed by the UCLA Technology Development Group on behalf of the Regents of the University of California.
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